Alterity Therapeutics

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     InvestorHub Global UI Overrides
     Prefix: ih-
     Purpose:
       1) Hide share price / ticker widget
       2) Hide survey launcher button
       3) Hide injected timed survey popup panel
     ========================================================================== */

  /* --------------------------------------------------------------------------
     1) Hide ASX ticker / share price widget
     -------------------------------------------------------------------------- */
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    display: none !important;
    visibility: hidden !important;
    width: 0 !important;
    height: 0 !important;
    overflow: hidden !important;
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  #share-price + .h-8.w-px.bg-hubs-dividers {
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  /* --------------------------------------------------------------------------
     2) Hide the floating survey launcher button (bottom-right)
     (your earlier snippet)
     -------------------------------------------------------------------------- */
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    display: none !important;
    visibility: hidden !important;
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  /* --------------------------------------------------------------------------
     3) Hide the timed/injected survey popup panel
     (the wrapper you pasted)
     -------------------------------------------------------------------------- */

  /* Extremely specific: matches the exact utility-class signature of that popup */
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    display: none !important;
    visibility: hidden !important;
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  /* Slightly more flexible fallback (in case spacing classes change later)
     Still specific enough to avoid collateral damage */
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    visibility: hidden !important;
    pointer-events: none !important;
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  /* Optional: if it ever mounts but stays in DOM, ensure it can't capture clicks */
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    pointer-events: none !important;
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ATH434

Parkinsonian Diseases

Multiple System Atrophy

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Montserrat

ATH434 Preserves Dopaminergic Neurons, Reduces α-synuclein Oligomerization, and Improves Motor Function in a Transgenic Murine Multiple System Atrophy Model (1842)

American Neurological Association (ANA): ATH434 Preserves Dopaminergic Neurons, Reduces α-synuclein Oligomerization, and Improves Motor Function in a Transgenic Murine Multiple System Atrophy Model